Cases 1 and 4 highlight the role of MCS independent of revascularization.
Repeat cardiac angiography revealed patent stents and no propagation of the dissection, suggesting that the mechanism of cardiogenic shock was not ongoing ischemia.
ventricular dysfunction and cardiogenic shock without ongoing ischemia
The etiology of left ventricular dysfunction and shock in the absence of ischemia in SCAD patients is unknown.
catecholamine surges, coronary artery wall stress, and a hyperdynamic myocardium
avoid a risky revascularization procedure and instead support the myocardium while allowing SCAD vessels to heal on their own.
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